puerperal thrombosis - definitie. Wat is puerperal thrombosis
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Wat (wie) is puerperal thrombosis - definitie

THREE FACTORS THOUGHT TO CONTRIBUTE TO THROMBOSIS
Thrombosis triad

Postpartum disorder         
HUMAN DISEASE
Puerperal disorders; Puerperal disorder
A postpartum disorder or puerperal disorder is a disease or condition which presents primarily during the days and weeks after childbirth called the postpartum period. The postpartum period can be divided into three distinct stages: the initial or acute phase, 6–12 hours after childbirth; subacute postpartum period, which lasts two to six weeks, and the delayed postpartum period, which can last up to six months.
Cerebral venous sinus thrombosis         
  • A dural venous sinus thrombosis of the transverse sinus. Greater on the right than left.
  • CT venogram showing a filling defect in the sagittal sinus (black arrow)
  • blood outflow from cerebral veins]] to the [[internal jugular vein]] at the [[base of skull]]
PRESENCE OF ACUTE THROMBOSIS (A BLOOD CLOT) IN THE DURAL VENOUS SINUSES, WHICH DRAIN BLOOD FROM THE BRAIN.
Nonpyogenic thrombosis of intracranial venous system; Cerebral venous thrombosis; Dural sinus thrombosis; Cerebral sinus thrombosis; Sagittal sinus thrombosis; Superior sagittal sinus thrombosis; Cerebral vein thrombosis; CVST; Dural venous sinus thrombosis; Venous sinus thrombosis; Nonpyogenic thrombosis of intracranial venous sinus; Marantic thrombosis; Transverse sinus thrombosis; Intracranial venous thrombosis
Cerebral venous sinus thrombosis (CVST), cerebral venous and sinus thrombosis or cerebral venous thrombosis (CVT), is the presence of a blood clot in the dural venous sinuses (which drain blood from the brain), the cerebral veins, or both. Symptoms may include severe headache, visual symptoms, any of the symptoms of stroke such as weakness of the face and limbs on one side of the body, and seizures, which occur in around 40% of patients.
deep-vein thrombosis         
  • alt=An image showing major arm veins
  • 500px
  • alt=Artistic rendering of DVT
  • alt=Imagine showing the coagulation, which includes a group of proteins that regulate clots. DVT risk can be altered by abnormalities in the cascade.
  • alt=Image of a leg with "pitting" edema, a transient depression of the skin after pressure is applied. When this happens on one side, it increases the likelihood of DVT.
  • alt=Image depicting D-dimer production
  • alt=Drawing showing that moving down the body, the inferior vena cava branches into 2 common iliac veins. The common iliac veins split into the internal iliac and external iliac veins. The external iliac veins give rise to the common femoral veins.
  • An IVC filter
  • The incision for a completed [[knee replacement surgery]], a procedure that can precipitate DVT formation
  • Rudolf Virchow
  • [[Serena Williams]] has spoken at length about a frightening encounter she had with VTE while she was hospitalized in 2017.<ref name=Serena/>
  • An example of a compression stocking
  • Warfarin, a common vitamin K antagonist, was the mainstay of pharmacological treatment for about 50 years.
FORMATION OF A BLOOD CLOT (THROMBUS) IN A DEEP VEIN
DVT; Traveller's thrombosis; Deep-vein thrombosis; Deep Vein Thrombosis; Deep venous thrombosis; Deep-vein thombosis; Deep Vein Thombosis; Coach flying syndrome; Economy class syndrome; Traveller's Thrombosis; Deep vein obstruction; Deep vein thrombosis in pregnancy; Iliac vein thrombosis; Deep vein thrombosis in lower limb (NOS); Blood clot in the leg; Leg blood clot; Dutch Primary Care Rule; Proximal DVT; Distal DVT; Iliofemoral DVT
¦ noun thrombosis in a vein lying deep below the skin, especially in the legs.

Wikipedia

Virchow's triad

Virchow's triad or the triad of Virchow () describes the three broad categories of factors that are thought to contribute to thrombosis.

  • Hypercoagulability
  • Hemodynamic changes (stasis, turbulence)
  • Endothelial injury/dysfunction

It is named after the renowned German physician Rudolf Virchow (1821-1902). However, the elements comprising Virchow's triad were not proposed by Virchow. Neither did he ever suggest a triad to describe the pathogenesis of venous thrombosis. In fact, it was not until decades after Virchow's death that a consensus was reached proposing that thrombosis is the result of alterations in blood flow, vascular endothelial injury, or alterations in the constitution of the blood. Still, the modern understanding of the factors leading to embolism is similar to the description provided by Virchow. Virchow's triad remains a useful concept for clinicians and pathologists alike in understanding the contributors to thrombosis.